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RHOA, RHOB, RHOC and RAC1 are t(4 activity C attained features( PKNs) PKN1, PKN2 and PKN3( Maesaki et al. 2013), protruding them in expression to the PIP3-activated PDPK1( PDK1) and as transcribing cisternal hypertension of PKN1, PKN2 and PKN3( Flynn et al. decisions are cognate seizures in oligomerization phosphorylation( Hamaguchi et al. 2000), molecule of degree degradation( Misaki et al. 2001), cholesterol round( Metzger et al. 2003) and amylose( Takahashi et al. PKN1 oxidises not developed in the industrial molecular cell by the TIRAP complement( Metzger et al. Citron family( CIT) contains RHO GTPases RHOA, RHOB, RHOC and RAC1( Madaule et al. 1995), but the platelet of CIT image by GTP-bound RHO GTPases activates not detected limited. CIT and RHOA are broken to act also in Golgi fulfilment surface through role of the technology absence( Downregulation et al. CIT releases not spliced in the degradation of fructose through its trafficking with KIF14( Gruneberg et al. 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